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Study Finds Porn Exposure Associated With Smaller Brain Region
New submitter Bodhammer (559311) writes "German researchers looked at the brains of 64 men between the ages of 21 and 45 and found that one brain region (the striatum, linked to reward processing), was smaller in the brains of porn watchers, and that a specific part of the same region is also less activated when exposed to more pornography." While it's tempting to cast blame, "the study doesn't confirm whether watching porn causes the changes, or whether people with a certain brain type are inherently more apt to tune into X-rated content." The study's abstract is available; the paper itself is pay-walled.
At last a study that concerns /.ers more than any of the other stuff to be found here lately.
Almost certainly.
Don't worry though, the reason you'll prosper if you live long is because aging gradually erodes the hold your base urges have upon you.
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Where did they find them?
Help stamp out iliturcy.
Seeing as we have no evidence that such an area being smaller is a good or a bad thing I would caution away from using this information as anything more than an observation.
In other words, people with a smaller brain region are less capable of speaking the truth.
The creatures outside looked from Alt-Right to Antifa; but already it was impossible to say which was which.
No, just that particular region. Can a brain region be measured in negative area?
Do not look into laser with remaining eye.
So all guys have a smaller region of the brain?
More pseudoscience. They say that they're not sure whether this means that porn shrinks your brain, or if the shrunken brain causes porn viewing. But, this leaves out the very real possibility that this correlation means nothing whatsoever. The site below collects correlations that look pretty convincing in the graphs, but quite obviously are unlikely to be cases of causation in either direction:
http://www.tylervigen.com/
This is an interesting question to research but I wish TFA wasn't paywalled b/c there are several factors they'd need to address that aren't mentioned in the description.
1. is this just *watching* porn or watching and fapping...also fapping w/o any physical media stimuli
2. they'd need to compare a control test with a completely different behavior/stimuli that triggers those same parts of the brain...it's "reward center" so maybe something with video games or receiving compliments on your appearance
3. what about actual sex? does it do the same thing to this part of the brain? I know /.'ers probably can't imagine this but it's possible to have sex too much so I'd definitey need to see if actual sex is any different than their results with porn.
those are some good starters...plenty of room for further research which means job security ;)
Thank you Dave Raggett
Only by these guys http://www.xxxchurch.com/.
The striatum is implicated in ADHD and several studies have indicated reduced grey matter volume in that region for ADHD sufferers. Failure in the dopamine pathways will generally cause engagement in dopamine releasing activities, as a method of self medication.
So it's not like finding a correlation between dopamine seeking and striatum deficiencies is unexpected. And the most likely direction of causation is that the deficient reward region causes the increased porn watching.
Frankly I find the gleeful reporting on the issue to be somewhat offensive. Insinuating that what is probably an inherent handicap is something the handicapped did to themselves by being 'immoral' is quite disgusting.
Well, one forearm anyway.
What a fool believes, he sees, no wise man has the power to reason away.
Once you jack off to Japanese girls puking in each other's mouths, you can't exactly go back to Playboy.
The portion of the brain they are talking about is turned on by watching rom-com's.
Sleep your way to a whiter smile...date a dentist!
Alcoholism would be among the least useful compulsive behaviors to compare because a large percentage of alcoholics metabolize alcohol differently than non-alcoholics. Therefore a significant portion of the problem is purely physical, not behavioral or centered in the brain. Potheads might be a better comparison - as far as we know, everyone's body processes THC in essentially the same way. There are of course many other compulsive behaviors that seem to be purely psychological / behavioral / brain-based, as opposed to being caused in part by differences in other bodily systems.
Specifically, we know that the difference in alcohol processing is not a RESULT of drinking because non-drinkers who were siblings of alcoholics often displayed the same trait. It appears likely that these siblings were genetically predisposed to become alcoholics, but had not activated the condition by introducing alcohol into their system. The metabolic difference happens after alcohol turns into acetaldehyde. Most people have TWO enzymes that quickly break down the acetaldehyde. Many alcoholics are missing one of the two enzymes, which is controlled by a certain gene that has been mapped. With one of the enzymes missing, the acetaldehyde remains for a much longer period of time. During the period that acetaldehyde is present in significant amounts, the alcoholic experiences the phenomenon of craving - an overpowering desire for more alcohol. Therefore, it seems that alcoholism is largely caused by the lack of an important enzyme, rather than a difference in brain function.
Of course, if a person who is missing the enzyme never becomes intoxicated in the first place, the craving will not be triggered. Also, there are many people who drink excessively but do not lack the enzyme and therefore probably do not have the craving effect. There are of course behavioral and psychological factors involved for these people, who could be called "hard drinkers".
The truth is that the brain region is used for lying, it is smaller in men who admit to watching porn and larger for men who lie about it.
In women it is about the size of a coconut.
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You may solo them, I prefer them in a group.
That may call into question cause, but not consequence; if pursuing a drug addiction causes behavioural changes (which alcoholism certainly does, even when sober) then it is not unreasonable to assume some alteration in activity or structure of the striatum comes as a result of this. The paper cites previous work on the topic, saying that alcoholism (and drug addiction in general) is indeed correlated with changes in the striatum, so I wouldn't really question that part of it too vigorously. My complaint is basically that they seem to be violating a triangle inequality: the brain change is tightly correlated with two addictive behaviours in the sample, but they're only weakly correlated with each other. It sounds to me like porn is correlated with novel changes in the same region.
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Consider another well-known disease that involves a missing compound needed for metabolism - diabetes. Supplying insulin helps a diabetic patient, but doesn't "cure" the disease.
It certainly seems plausible that by identifying people who lack the enzyme and either a) providing the enzyme or b) warning them of the problem, many cases could be avoided. People who lack the enzyme but abstain from alcohol seem to be okay generally, so that would seem a reasonable strategy. However, digestion of food creates alcohol, so that's an area where further study may be needed.
The other thing is, once you take care of the enzyme in an active alcoholic, you're left with just a regular drug addict - alcohol is a drug, of course. You've treated the thing that makes alcohol addicts different from other addicts, but they're still an addict. We know also that alcoholism includes some positive feedback cycles. People often have a drink when negative events happens in their lives. When they drink excessively, that causes more negative events. The alcoholic typically ends up in a cycle of dependency. Indeed, it seems that many people identified as alcoholic have only the psychological dependance and are not lacking the indicated enzyme. Enzyme therapy therefore wouldn't be expected to work in these "type II" drinkers. It may turn "type I" (enzymatic alcoholics) into type II (psychologically dependent), but that doesn't seem to be a huge win. Enzyme therapy would probably need to be combined with treatment for the psychological side as well
Looking at it another way, there are two primary issues with alcoholics. A) when they drink, they can't stop and B) they start drinking, even given the knowledge of B. Treating A doesn't fix the odd metal obsession that we see manifested in B.