The USB port on sony Video Cameras is for transferring still images off the compactflash card, while the firewire port is used for transferring DV (Digital Video) content. If your mac does not have a firewire port, i highly doubt it would be running OS X. Get a 6-to-4 pin firewire cable to hook the camera up and try iMovie.
When apple upgrades their bottom line, the professional line is never too far away. I remember a while ago when Powerbook G3's were all the rage, apple created the G3 iBook and then immediately moved the powerbooks to a G4 Chip (and the Titanium enclosure). The same thing happened with The iMacs and the G5's... when iMacs + eMacs became G4's (the current models), the G4 towers were soon phased out and replaced with G5's... the pro powerbooks will be G5-based soon.
According to ther site, the guy uses three firewire-to-ide interface cards and a firewire hub. Obviously, from the screenshot, he uses Mac OS X, so I assume has has to be using software RAID... I dont see any mention of a RAID controller or some sort of combinatorial device other than the firewire hub, and that cetainly will not combine drives.
FYI, Mac OS X includes software RAID by default, it's accessible in Disk Utility, located in/Applications/Utilities
Am I the only one who noticed that the woman in the BBC Article's picture (directly above the "The MSBlast worm hit some users hard" Caption text) is using an old mac, and therefore, is not struggling with the MSBlast worm?
The power button and display/contrast knobs on the side of the monitor give it away....
Also, from the article: "But viruses that take advantage of new found flaws in the chunk of computer code exploited by MSBlast look set to arrive even sooner." -- Does this mean that even though microsoft cleaned up the code that was used by MSBlast as a backdoor, they still overlooked some code in the same region?
a correction
on
Google Turns 5
·
· Score: 4, Informative
the article states 200 million queries a day, not 200,000 million (200 billion) queries as the slashdot post says.
Macs pretty much dominate the Biochem, Biotech and Bioinformatics markets, and are used for most tasks such as coordinating Real-time PCR and sequencing equipment, to flow cytometry, to molecular modeling and advanced chemistry.
Not to mention that the CEO of Genentech (Arthur Levinson) is on Apple's board of directors, and that NCBI BLAST (Basic Local Alignment Search Tool -- a best-fit "search engine" for the genome, if you will) run amazingly, screamingly fast on Altivec-enabled machines.
the details of Apple/Genentech BLAST are available here
Ambrosia has taken a terrific step by porting override and EV classic to the Nova engine.
After downloading them, i realized that the ported version has a few fatal flaws which detract much from the original:
a) no forklift through conventional cheat -- the forklift was a terriffic weapon, accessed by option clicking the logo on the title screen, witing for a certail line of text to appear, and holding control-option-command-shift. This doesnt work under the nova engine.
b) no EV-Edit/.rsrc types for the Nova pilots! After playing the game a few times, it became repetitive; I got most of my play time out of the game by modifying it... making my own ships, planets, weapons, and outfits. the customizability of EV was endless and made the game fun for a long time... The ported versions save Nova-type pilot files which have to editing tools yet.
Although I'm enthused about the new releases, there's still a large part of the original EVs missing for me
Would you believe that some crazy individuals out there use computers not primarily for gaming? They actually like to get work done (a prepostorous concept!), without their operating system getting in their way (those rogues!).
Some of us developers also like to code more important applications than Neverwinter Nights Modules, in which case, hey look!, a mac is a terrific choice, not only for it's power and speed, but also for productivity applications, it's teriffic cocoa (obj-c/java) APIs, and included IDE!
First of all, it's beta 2 and it's not slow at all.
Actually, it's been beta 3 for a few months now, and can be had at http://www.apple.com/macosx/x11/. If you get it, make sure to get the SDK too, so that you can compile new X11 apps (such as those provided by fink)
I also saw another DVD upgrade for the PowerBook, but it doesn't mention running iDVD, which would be one of the requirements for me actually upgrading.
And I quote, directly from the linked website (http://store.powerbook1.com/mceindvsupup.html)... Burn DVDs (at 2X) and CDs anywhere you can take your PowerBook... (And, it's compatible with iDVD!)
All you people do is whine and whine about languages!
Back in my day, I had a bunch of OR and NOT gates and some solder. When I was very good, my parents would buy me an AND gate for my birthday. Those were the days.
That's the COMMAND key. If you've ever cut & pasted on a mac using the keyboard, or used almost any keyboard shortcuts, then you definetly have a use for "that key"
can anyone direct me to an example of how WebCore is implemented?
I am trying to make a simple WWWView (its a subclass of NSView) in an Objective-C/Cocoa application I am building, and it seems that WebCore is just what I need to use.
The header files havent helped me much, as im not sure quite what the methods are to make the View a WebCore view.
I believe the correct class to use from the WebCoreFramework is WebCoreBridge, but when i do [[WebCoreBridge alloc] init] and assign the result to a WebCoreBridge *, the app dies with SIGTRAP.
Basically, I'm looking for help in getting a WebCore view in the GUI of an application i'm writing.
Right now Im sort of stuck.
if anyone knows how to implement the WebCore, please reply or email me (madcoder@madcoder.net)
By the same reasoning (blocking the images is "theft"), anyone could argue that Lynx or any other text-based browser is just a tool for theft, as of course the images won't be shown.
The problem with p53 is that cancer cells contain a mutated p53 gene.
Normal nonmutated p53 acts as a tumor suppressor, binding to genomic DNA and starting gene expression of p21 if the DNA copy is incorrect (as is the case with cancerous cells with genomic mutations).
p21 halts the cell division, stopping reproduction of the cancerous cell.
In a already cancerous existing cell with a p53 mutation, the cell will divide normally, because the p53 is mutated and it cannot bind DNA effectively. Therefore, the p21 will not get signalled, and the cancer will continue to multiply.
It's not all your cells dying, which is the only reason you are wrong.
Different cells have different cell-surface receptors. Some receptors create apoptotic signals within the cell, others don't. It is a direct result of external happenings within your body... i.e. a T-Cell binds to a cell with a nonself antigen which also expresses Fas Ligand. Only THAT cell will die.
Not massive cell death, death of certain targeted cells bearing certain apoptotic receptors.
Sorry abut the raw-HTML post above. I forgot to switch from code mode. Here is the correct version: Because it is an interesting and often misunderstood subject, here is a small primer on the topic of apoptosis (Programmed Cell Death). FYI I work in an immuno lab which uses apoptosis as a main treatment for transplant tolerance.
Death by suicide
Cells that are induced to commit suicide:
shrink
have their mitochondria break down with the release of cytochrome c
develop bubble-like blebs on their surface
have the chromatin (DNA and protein) in their nucleus degraded
break into small, membrane-wrapped, fragments
The phospholipid phosphatidylserine, which is normally hidden within the plasma membrane is exposed on the surface.
This is bound by receptors on phagocytic cells like and dendritic cells which then engulf the cell fragments.
The phagocytic cells secrete cytokines that inhibit inflammation.
The pattern of events in death by suicide is so orderly that the process is often called programmed cell death or PCD. The cellular machinery of programmed cell death turns out to be as intrinsic to the cell as, say, mitosis.
Why should a cell commit suicide?
There are two different reasons.
1. Programmed cell death is as needed for proper development as mitosis is.
Examples:
The resorption of the tadpole tail at the time of its metamorphosis into a frog occurs by apoptosis.
The formation of the fingers and toes of the fetus requires the removal, by apoptosis, of the tissue between them.
The sloughing off of the inner lining of the uterus (the endometrium) at the start of menstruation occurs by apoptosis.
The formation of the proper connections (synapses) between neurons in the brain requires that surplus cells be eliminated by apoptosis
2. Programmed cell death is needed to destroy cells that represent a threat to the integrity of the organism.
Examples:
Cells infected with viruses One of the methods by which cytotoxic T lymphocytes (CTLs) kill virus-infected cells is by inducing apoptosis. (And some viruses mount countermeasures to thwart it.) Cells with DNA damage Damage to its genome can cause a cell
to disrupt proper embryonic development leading to birth defects
to become cancerous.
Cells respond to DNA damage by increasing their production of p53. p53 is a potent causer of apoptosis. Is it any wonder that mutations in the p53 gene, producing a defective protein, are so often found in cancer cells (that represent a lethal threat to the organism if permitted to live)?
Cancer cells Radiation and chemicals used in cancer therapy induce apoptosis in some types of cancer cells.
<p>Because it is an interesting and often misunderstood subject, here is a small primer on the topic of apoptosis (Programmed Cell Death). <br> FYI I work in an immuno lab which uses apoptosis as a main treatment for transplant tolerance.</p> <p><strong><em>Deat h by suicide</em></strong></p> <p>Cells that are induced to commit suicide: </p> <ul> <li>shrink <li>have their mitochondria break down with the release of cytochrome c <li>develop bubble-like blebs on their surface <li>have the chromatin (DNA and protein) in their nucleus degraded <li>break into small, membrane-wrapped, fragments <li>The phospholipid phosphatidylserine, which is normally hidden within the plasma membrane is exposed on the surface. <li>This is bound by receptors on phagocytic cells like and dendritic cells which then engulf the cell fragments. <li>The phagocytic cells secrete cytokines that <b>inhibit inflammation</b>. </ul> <p>The pattern of events in death by suicide is so orderly that the process is often called <b>programmed cell death</b> or <b>PCD</b>. The cellular machinery of programmed cell death turns out to be as intrinsic to the cell as, say, mitosis. </p> <p>Why should a cell commit suicide?</p> <p>There are two different reasons. </p> <p><strong>1. Programmed cell death is as needed for proper development as mitosis is.</strong></p> <blockquote> <p><em>Examples: </em></p> <ul> <li>The resorption of the tadpole tail at the time of its metamorphosis into a frog occurs by apoptosis. </li> <li>The formation of the fingers and toes of the fetus requires the removal, by apoptosis, of the tissue between them. </li> <li>The sloughing off of the inner lining of the uterus (the endometrium) at the start of menstruation occurs by apoptosis. </li> <li>The formation of the proper connections (synapses) between neurons in the brain requires that surplus cells be eliminated by apoptosis</li> </ul> </blockquote> <p><strong>2. Programmed cell death is needed to destroy cells that represent a threat to the integrity of the organism.</strong></p> <blockquote> ; <p><em>Examples:</em> </p> <dl> <dl> <dt><b>Cells infected with viruses</b> </dt> <dd>One of the methods by which <b>cytotoxic T lymphocytes</b> (CTLs) kill virus-infected cells is by inducing apoptosis. (And some viruses mount countermeasures to thwart it.) </dd> <dt><b>Cells with DNA damage</b></dt> <dd>Damage to its genome can cause a cell <ul> <li>to disrupt proper embryonic development leading to birth defects </li> <li>to become cancerous.</li> </ul> <p>Cells respond to DNA damage by increasing their production of p53. p53 is a potent causer of apoptosis. Is it any wonder that mutations in the p53 gene, producing a defective protein, are so often found in cancer cells (that represent a lethal threat to the organism if permitted to live)? </p> </dd> <dt><b>Cancer cells</b></dt> <dd>Radiation and chemicals used in cancer therapy induce apoptosis in some types of cancer cells.</dd> </dl> </dl> </blockquote>
Slashdot Mirror
The USB port on sony Video Cameras is for transferring still images off the compactflash card, while the firewire port is used for transferring DV (Digital Video) content. If your mac does not have a firewire port, i highly doubt it would be running OS X. Get a 6-to-4 pin firewire cable to hook the camera up and try iMovie.
If standing in a bathtub with an electric iron isn't your cup of tea, here's a guiltless Google Link.
When apple upgrades their bottom line, the professional line is never too far away. I remember a while ago when Powerbook G3's were all the rage, apple created the G3 iBook and then immediately moved the powerbooks to a G4 Chip (and the Titanium enclosure). The same thing happened with The iMacs and the G5's... when iMacs + eMacs became G4's (the current models), the G4 towers were soon phased out and replaced with G5's... the pro powerbooks will be G5-based soon.
According to ther site, the guy uses three firewire-to-ide interface cards and a firewire hub. Obviously, from the screenshot, he uses Mac OS X, so I assume has has to be using software RAID... I dont see any mention of a RAID controller or some sort of combinatorial device other than the firewire hub, and that cetainly will not combine drives.
/Applications/Utilities
FYI, Mac OS X includes software RAID by default, it's accessible in Disk Utility, located in
In other news, the KGB has filed a lawsuit against the Kryptos Group under the DMCA, claiming that their IP has now been stolen.
The sad part of this is that in today's world somrthing similar could happen.
Am I the only one who noticed that the woman in the BBC Article's picture (directly above the "The MSBlast worm hit some users hard" Caption text) is using an old mac, and therefore, is not struggling with the MSBlast worm?
The power button and display/contrast knobs on the side of the monitor give it away....
Also, from the article: "But viruses that take advantage of new found flaws in the chunk of computer code exploited by MSBlast look set to arrive even sooner." -- Does this mean that even though microsoft cleaned up the code that was used by MSBlast as a backdoor, they still overlooked some code in the same region?
the article states 200 million queries a day, not 200,000 million (200 billion) queries as the slashdot post says.
Macs pretty much dominate the Biochem, Biotech and Bioinformatics markets, and are used for most tasks such as coordinating Real-time PCR and sequencing equipment, to flow cytometry, to molecular modeling and advanced chemistry.
Not to mention that the CEO of Genentech (Arthur Levinson) is on Apple's board of directors, and that NCBI BLAST (Basic Local Alignment Search Tool -- a best-fit "search engine" for the genome, if you will) run amazingly, screamingly fast on Altivec-enabled machines.
the details of Apple/Genentech BLAST are available here
Ambrosia has taken a terrific step by porting override and EV classic to the Nova engine.
After downloading them, i realized that the ported version has a few fatal flaws which detract much from the original:
a) no forklift through conventional cheat -- the forklift was a terriffic weapon, accessed by option clicking the logo on the title screen, witing for a certail line of text to appear, and holding control-option-command-shift. This doesnt work under the nova engine.
b) no EV-Edit/.rsrc types for the Nova pilots! After playing the game a few times, it became repetitive; I got most of my play time out of the game by modifying it... making my own ships, planets, weapons, and outfits. the customizability of EV was endless and made the game fun for a long time... The ported versions save Nova-type pilot files which have to editing tools yet.
Although I'm enthused about the new releases, there's still a large part of the original EVs missing for me
Would you believe that some crazy individuals out there use computers not primarily for gaming? They actually like to get work done (a prepostorous concept!), without their operating system getting in their way (those rogues!).
Some of us developers also like to code more important applications than Neverwinter Nights Modules, in which case, hey look!, a mac is a terrific choice, not only for it's power and speed, but also for productivity applications, it's teriffic cocoa (obj-c/java) APIs, and included IDE!
First of all, it's beta 2 and it's not slow at all.
Actually, it's been beta 3 for a few months now, and can be had at http://www.apple.com/macosx/x11/. If you get it, make sure to get the SDK too, so that you can compile new X11 apps (such as those provided by fink)
Apparently Intel follows an all-to-common business model, which has been scientifically proven to inevitably lead to cannibalism:
I also saw another DVD upgrade for the PowerBook, but it doesn't mention running iDVD, which would be one of the requirements for me actually upgrading.
...
And I quote, directly from the linked website (http://store.powerbook1.com/mceindvsupup.html)
Burn DVDs (at 2X) and CDs anywhere you can take your PowerBook... (And, it's compatible with iDVD!)
Bah!
All you people do is whine and whine about languages!
Back in my day, I had a bunch of OR and NOT gates and some solder. When I was very good, my parents would buy me an AND gate for my birthday. Those were the days.
Not to mention easy access to sensitive information in emails, documents, and PIMs that the user currently is running and are resident in memory.
1. Cow Manure
2. ?
3. Energy!
That's the COMMAND key. If you've ever cut & pasted on a mac using the keyboard, or used almost any keyboard shortcuts, then you definetly have a use for "that key"
can anyone direct me to an example of how WebCore is implemented?
I am trying to make a simple WWWView (its a subclass of NSView) in an Objective-C/Cocoa application I am building, and it seems that WebCore is just what I need to use.
The header files havent helped me much, as im not sure quite what the methods are to make the View a WebCore view.
I believe the correct class to use from the WebCoreFramework is WebCoreBridge, but when i do [[WebCoreBridge alloc] init] and assign the result to a WebCoreBridge *, the app dies with SIGTRAP.
Basically, I'm looking for help in getting a WebCore view in the GUI of an application i'm writing.
Right now Im sort of stuck.
if anyone knows how to implement the WebCore, please reply or email me (madcoder@madcoder.net)
imagine a beowulf cluster of those....
By the same reasoning (blocking the images is "theft"), anyone could argue that Lynx or any other text-based browser is just a tool for theft, as of course the images won't be shown.
Did anyone else get this strange (yet true) response after blocking the popup? (click link before you mod me down!!)
The problem with p53 is that cancer cells contain a mutated p53 gene.
.
Normal nonmutated p53 acts as a tumor suppressor, binding to genomic DNA and starting gene expression of p21 if the DNA copy is incorrect (as is the case with cancerous cells with genomic mutations)
p21 halts the cell division, stopping reproduction of the cancerous cell.
In a already cancerous existing cell with a p53 mutation, the cell will divide normally, because the p53 is mutated and it cannot bind DNA effectively. Therefore, the p21 will not get signalled, and the cancer will continue to multiply.
It's not all your cells dying, which is the only reason you are wrong.
Different cells have different cell-surface receptors. Some receptors create apoptotic signals within the cell, others don't. It is a direct result of external happenings within your body... i.e. a T-Cell binds to a cell with a nonself antigen which also expresses Fas Ligand. Only THAT cell will die.
Not massive cell death, death of certain targeted cells bearing certain apoptotic receptors.
Sorry abut the raw-HTML post above. I forgot to switch from code mode. Here is the correct version:
Because it is an interesting and often misunderstood subject, here is a small primer on the topic of apoptosis (Programmed Cell Death).
FYI I work in an immuno lab which uses apoptosis as a main treatment for transplant tolerance.
Death by suicide
Cells that are induced to commit suicide:
The pattern of events in death by suicide is so orderly that the process is often called programmed cell death or PCD. The cellular machinery of programmed cell death turns out to be as intrinsic to the cell as, say, mitosis.
Why should a cell commit suicide?
There are two different reasons.
1. Programmed cell death is as needed for proper development as mitosis is.
2. Programmed cell death is needed to destroy cells that represent a threat to the integrity of the organism.
<p>Because it is an interesting and often misunderstood subject, here is a small
primer on the topic of apoptosis (Programmed Cell Death). <br>
FYI I work in an immuno lab which uses apoptosis as a main treatment for transplant
tolerance.</p>
<p><strong><em>Deat h by suicide</em></strong></p>
<p>Cells that are induced to commit suicide: </p>
<ul>
<li>shrink
<li>have their mitochondria break down with the release of cytochrome c
<li>develop bubble-like blebs on their surface
<li>have the chromatin (DNA and protein) in their nucleus degraded
<li>break into small, membrane-wrapped, fragments
<li>The phospholipid phosphatidylserine, which is normally hidden within the
plasma membrane is exposed on the surface.
<li>This is bound by receptors on phagocytic cells like and dendritic cells
which then engulf the cell fragments.
<li>The phagocytic cells secrete cytokines that <b>inhibit inflammation</b>.
</ul>
<p>The pattern of events in death by suicide is so orderly that the process is
often called <b>programmed cell death</b> or <b>PCD</b>. The cellular machinery
of programmed cell death turns out to be as intrinsic to the cell as, say, mitosis.
</p>
<p>Why should a cell commit suicide?</p>
<p>There are two different reasons. </p>
<p><strong>1. Programmed cell death is as needed for proper development as mitosis
is.</strong></p>
<blockquote>
<p><em>Examples: </em></p>
<ul>
<li>The resorption of the tadpole tail at the time of its metamorphosis into
a frog occurs by apoptosis. </li>
<li>The formation of the fingers and toes of the fetus requires the removal,
by apoptosis, of the tissue between them. </li>
<li>The sloughing off of the inner lining of the uterus (the endometrium)
at the start of menstruation occurs by apoptosis. </li>
<li>The formation of the proper connections (synapses) between neurons in
the brain requires that surplus cells be eliminated by apoptosis</li>
</ul>
</blockquote>
<p><strong>2. Programmed cell death is needed to destroy cells that represent
a threat to the integrity of the organism.</strong></p>
<blockquote> ;
<p><em>Examples:</em> </p>
<dl>
<dl>
<dt><b>Cells infected with viruses</b> </dt>
<dd>One of the methods by which <b>cytotoxic T lymphocytes</b> (CTLs) kill
virus-infected cells is by inducing apoptosis. (And some viruses mount
countermeasures to thwart it.) </dd>
<dt><b>Cells with DNA damage</b></dt>
<dd>Damage to its genome can cause a cell
<ul>
<li>to disrupt proper embryonic development leading to birth defects
</li>
<li>to become cancerous.</li>
</ul>
<p>Cells respond to DNA damage by increasing their production of p53.
p53 is a potent causer of apoptosis. Is it any wonder that mutations
in the p53 gene, producing a defective protein, are so often found in
cancer cells (that represent a lethal threat to the organism if permitted
to live)? </p>
</dd>
<dt><b>Cancer cells</b></dt>
<dd>Radiation and chemicals used in cancer therapy induce apoptosis in some
types of cancer cells.</dd>
</dl>
</dl>
</blockquote>